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Experimental anti-cytokine treatments—including agents that block these cytokines and their signaling cascades (such as NF-κB and MAPK pathways)—are being studied for their potential to dampen inflammation and decelerate degenerative changes [35]

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Angiotensin AT2 receptor stimulation is anti-inflammatory in lipopolysaccharide-activated THP-1 macrophages via increased interleukin-10 production