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Because Pin1 has been implicated in dephosphorylation of tau protein, it can be hypothesized that in vivo oxidative modification of Pin1, as found in AD hippocampus, reduces Pin1 activity, leading to increased tau phosphorylation (199)

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Of significance, the reversible glutathionylation of cellular peptides and proteins is increasingly recognized as major cell signaling and regulatory mechanism (Dalle-Donne et al., 2007