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Furthermore, we discuss the interplay between viral infections and host ferroptosis process, with a focus on the mechanism of how viruses exploit ferroptosis for its own replication

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This process can be also elicited modulating cellular sensitivity through the cystine/glutamate antiporter (system Xc-) suppression, acting on their intracellular concentrations, or reducing the activity of GLS2, the enzyme catalyzing the conversion of glutamine into glutamate [31]