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The two mechanisms are complementary rather than redundant and BPC-157’s growth factor receptor upregulation and cytoprotective signalling creating a cellular environment permissive to repair, while TB-500’s actin dynamics modulation and PI3K/Akt activation drive the cell migration, vascular ingrowth, and matrix remodelling that execute the repair process at the tissue level

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JAMA 323 , 2427–2429 (2020)